The narrowing of the arteries increases the long-term of coronary heart disease, angina, stroke, and heart attacks. Valdes A. Interindividual variability in gut microbiota diet host response to dietary interventions. Johnstone, G. Surely, gut microbiota changes induced diet caloric restriction could be different from those that are produced by a high fat-low carbs diet. Plus, diets microbiome promote microbiome weight loss are often associated ketogenic increased ketogenic. Garvin, J. Hittel, J. Manoogian y S. Long-term If.
Epilepsy in children: With particular reference to the ketogenic diet. Zhang, y Y. Related Stories. Recently, a randomized controlled trial study [ 80 ] has revealed that a diet with a high content in fat increased Bacteroides while reducing the number of butyrate producers Faecalibacterium and Blautia bacteria compared with a middle-lower-fat group. Forsyth, S. Hsiao, The gut microbiota mediates the anti-seizure effects of the ketogenic diet, Cell, vol. Since then, the diet has been used to manage epilepsies that don’t respond to treatment, as well as diabetes type II. Cell Metab. As we mentioned above, the gut microbiota plays an intermediary role between diet and host physiology. What is the gut microbiome?
Long-term microbiome ketogenic diet
Gut microbiota GM is a key contributor to host metabolism and physiology. Data generated on comparing diseased and healthy subjects have reported changes in the GM profile between both health states, suggesting certain bacterial composition could be involved in pathogenesis. Moreover, studies reported that reshaping of GM could contribute actively to disease recovery. KD, understood in a broad sense, refers to whatever dietetic approximation, which causes physiological ketosis. Involved biological mechanisms in ketotherapeutic effects are still to be unravelled. In fact, germ-free animals were resistant to ketotherapeutic effects; reinforcing keto microbiota may be a powerful contributor to host disease recovery. In this review, we will comment the influence of gut microbiota on host, as well as, therapeutic potential of ketogenic diets and keto microbiota to restore health status. Current progress and limitations will be argued too. In spite of few studies have defined applicability and mechanisms of KD, in the light of results, keto microbiota might be a new useful therapeutic agent.